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Gepubliceerd: 12 september 2022 (4 weken geleden)
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Mutations in the SLC17A9 gene, encoding a vacuolar proton-translocating ATPase, have been associated with leukodystrophy or suspected to cause a part of clinically isolated ichthyosis. We describe a French cohort of three boys and four girls with a previously unreported phenotype of “atypical” leukodystrophy, characterized by rapidly progressive psychomotor regression, associated with cerebellar dysmyelination and hypomyelination of the brainstem. Four of the patients carry the p.N62S/p.N298S heterozygous mutations in SLC17A9, and a further patient carries a novel compound heterozygous SLC17A9 mutation. The observation of this novel phenotype in a European patient cohort highlights the importance of SLC17A9 mutations as a cause of leukodystrophy and demonstrates the need for broadening the phenotype of leukodystrophies by also taking into account less typical presentations of leukodystrophy. – 160 = 0 for z.
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